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This clinical entity has long been known to the medical profession. Gelineau gave it the name narcolepsy in 1880, although several authors had described the recurring attacks of irresistible sleep even before that time. Gelineau had also mentioned that the sleep attacks were sometimes accompanied by falls ( astasias ), but it was probably Loewenfeld (1902) who rst recognized the common association between the sleep attacks and the temporary paralysis of the somatic musculature during bouts of laughter, anger, and other emotional states; this was referred to as cataplectic inhibition by Henneberg (1916) and later as cataplexy by Adie (1926). The term sleep paralysis used to designate the brief, episodic loss of voluntary movement that occurs during the period of falling asleep (hypnagogic or predormital) or less often when awakening (hyp-

This is a fairly simple method that, in the end, returns a Boolean representing whether or not your database could be opened. You use the SQLiteOpenHelper created in your

You can see the results in Figure 9-2, where the text we sent back to the browser does appear in the browser. Nice.

2. Close the Layer Properties Manager palette. Notice that Objects now appears in the Layer text box on the Layers panel. 3. Save your work.

1. What are examples of controls 2. What is the purpose of the Toolbox 3. How do you add a control from the Toolbox on to your form

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as the Guillain-Barre syndrome, ruptured lumbar and cervical discs, multiple sclerosis, and lumbar and cervical spondylosis when the clinical data are uncertain. The central counterpart also pertains namely, that obliteration of the cortical waves (assuming that all preceding waves are unaltered) re ects profound damage to the somatosensory pathways in the hemisphere or to the cortex itself. As corollaries, the bilateral absence of cortical somatosensory waves after cardiac arrest is a powerful predictor of a poor clinical outcome; the persistent absence of a cortical potential after stroke usually indicates such profound damage that only a limited clinical recovery is to be expected. Evoked potential techniques have also been used in the experimental study of olfactory sensation (see Chap. 12).

2. Select the Items property and you'll notice three dots (. ..) appear alongside the word Collection. This is similar to what happens when you select the Items property for a drop-down list box.

signs CT scanning or MRI (which preferably should precede lumbar puncture) may be normal or abnormal A Subarachnoid hemorrhage from ruptured aneurysm, arteriovenous malformation, occasionally trauma (Chaps 34 and 35) B Acute bacterial meningitis (Chap 32) C Some forms of viral encephalitis (Chap 33) D Neoplastic and parasitic meningitides III Diseases that cause focal brainstem or lateralizing cerebral signs, with or without changes in the CSF CT scanning and MRI are usually abnormal A Hemispheral hemorrhage or massive infarction (Chap 34) B Brainstem infarction due to basilar artery thrombosis or embolism (Chap 34) C Brain abscess, subdural empyema, Herpes encephalitis (Chap 32) D Epidural and subdural hemorrhage and brain contusion (Chap 35) E Brain tumor (Chap 31) F Cerebellar and pontine hemorrhage G.

Miscellaneous: cortical vein thrombosis, some forms of viral encephalitis (herpes), focal embolic infarction due to bacterial endocarditis, acute hemorrhagic leukoencephalitis, disseminated (postinfectious) encephalomyelitis, intravascular lymphoma, thrombotic thrombocytopenic purpura, diffuse fat embolism, and others Using the clinical criteria outlined above, one can usually ascertain whether a given case of coma falls into one of these three categories Concerning the group without focal or lateralizing or meningeal signs (which includes most of the metabolic encephalopathies, intoxications, concussion, and postseizure states), it must be kept in mind that residua from previous neurologic disease may confuse the clinical picture Thus, an earlier hemiparesis from vascular disease or trauma may reassert itself in the course of uremic or hepatic coma with hypotension, hypoglycemia, diabetic acidosis, or following a seizure In hypertensive encephalopathy, focal signs may also be present.

Occasionally, for no understandable reason, one leg may seem to move less, one plantar re ex may be extensor, or seizures may be predominantly or entirely unilateral in a metabolic coma, particularly in the hyperglycemic-hyperosmolar states Babinski signs and extensor rigidity, conventionally considered to be indicators of structural disease, do sometimes occur in profound intoxications with a number of agents The diagnosis of concussion or of postictal coma depends on observation of the precipitating event or indirect evidence thereof Often a convulsive seizure is marked by a bitten tongue, urinary incontinence, and an elevated CK-skeletal muscle fraction; it may be followed by another seizure or burst of seizures The presence of small clonic or myoclonic convulsive movements of a hand or foot or uttering of the eyelids requires that an EEG be performed to determine whether status epilepticus is the cause of coma.

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